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In the past decade, clinical proteomics technologies, such as protein arrays and SELDI [54], are expected to provide new diagnostic markers that can translate into improved clinical testing

However, the viral infections also are related with diseases in the CNS characterized by the presence of leukocyte infiltration and inflammation, inducing a progressive damage [1], such as the progressive multifocal leukoencephalopathy with the John Cunningham virus (JC), AIDS-related dementia complex observed in HIV-1-infected patients, neurodevelopmental sequelae (mental retardation, cerebral palsy, and sensorineural hearing loss) caused by congenital cytomegalovirus (CMV) infection or cerebral mass lesions in immunocompromised adults CMV-infected, and HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP) that affects the human T-cell lymphotropic virus type 1 (HTLV-1)-infected individuals

Histones are well known to undergo a variety of post-translational modifications, namely phosphorylation, acetylation, methylation, and ubiquitination ( Figure 1 )

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